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dab colour-developing reagent  (Solarbio Inc)

 
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    Structured Review

    Solarbio Inc dab colour-developing reagent
    Main experimental reagents.
    Dab Colour Developing Reagent, supplied by Solarbio Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/dab colour-developing reagent/product/Solarbio Inc
    Average 90 stars, based on 1 article reviews
    dab colour-developing reagent - by Bioz Stars, 2026-04
    90/100 stars

    Images

    1) Product Images from "Expressions of ZNF436, β-catenin, EGFR, and CMTM5 in breast cancer and their clinical significances"

    Article Title: Expressions of ZNF436, β-catenin, EGFR, and CMTM5 in breast cancer and their clinical significances

    Journal: European Journal of Histochemistry : EJH

    doi: 10.4081/ejh.2021.3173

    Main experimental reagents.
    Figure Legend Snippet: Main experimental reagents.

    Techniques Used:



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    Image Search Results


    Main experimental reagents.

    Journal: European Journal of Histochemistry : EJH

    Article Title: Expressions of ZNF436, β-catenin, EGFR, and CMTM5 in breast cancer and their clinical significances

    doi: 10.4081/ejh.2021.3173

    Figure Lengend Snippet: Main experimental reagents.

    Article Snippet: DAB colour-developing reagent , DA1010 , Solarbio , China.

    Techniques:

    Main experimental reagents.

    Journal: European Journal of Histochemistry : EJH

    Article Title: Expressions of ZNF436, β-catenin, EGFR, and CMTM5 in breast cancer and their clinical significances

    doi: 10.4081/ejh.2021.3173

    Figure Lengend Snippet: Main experimental reagents.

    Article Snippet: DAB colour-developing reagent , DA1010 , Solarbio , China.

    Techniques:

    Decreased expression levels of BDNF and TrkB in the offspring rats’ hippocampus and the modulatory effects of DHF treatment. Immunohistochemistry staining revealed that the protein levels of BDNF and TrkB in the propofol exposure group (P group) were significant less than those in the control group (C group) ( A – C ). TrkB agonist (7,8‐ DHF ) reversed the expression of BDNF and TrkB. The protein levels of BDNF and TrkB in the 7,8‐ DHF treatment group ( PD group) were higher than those in the P group ( A – C ). Further, 7,8‐ DHF did not change the protein levels of BDNF and TrkB in rat offspring that had not been exposed to propofol ( CD group) ( A – C ). The changes of BDNF and TrkB mRNA expressions were similar to their proteins ( D ). ( A ) BDNF protein immunohistochemistry stain in offspring's hippocampus (400×); ( B ) TrkB protein immunohistochemistry stain in offspring's hippocampus (400×); ( C ) Data represents mean ± S.E. of 20 offspring rats in each group. ( D ) Real time PCR results of BDNF and TrkB mRNA expressions. Data represents mean ± S.E. of 20 offspring rats in each group, * P < 0.05.

    Journal: Journal of Cellular and Molecular Medicine

    Article Title: Propofol exposure during late stages of pregnancy impairs learning and memory in rat offspring via the BDNF ‐TrkB signalling pathway

    doi: 10.1111/jcmm.12884

    Figure Lengend Snippet: Decreased expression levels of BDNF and TrkB in the offspring rats’ hippocampus and the modulatory effects of DHF treatment. Immunohistochemistry staining revealed that the protein levels of BDNF and TrkB in the propofol exposure group (P group) were significant less than those in the control group (C group) ( A – C ). TrkB agonist (7,8‐ DHF ) reversed the expression of BDNF and TrkB. The protein levels of BDNF and TrkB in the 7,8‐ DHF treatment group ( PD group) were higher than those in the P group ( A – C ). Further, 7,8‐ DHF did not change the protein levels of BDNF and TrkB in rat offspring that had not been exposed to propofol ( CD group) ( A – C ). The changes of BDNF and TrkB mRNA expressions were similar to their proteins ( D ). ( A ) BDNF protein immunohistochemistry stain in offspring's hippocampus (400×); ( B ) TrkB protein immunohistochemistry stain in offspring's hippocampus (400×); ( C ) Data represents mean ± S.E. of 20 offspring rats in each group. ( D ) Real time PCR results of BDNF and TrkB mRNA expressions. Data represents mean ± S.E. of 20 offspring rats in each group, * P < 0.05.

    Article Snippet: The following main reagents were used in this study: propofol (Diprivan, AstraZeneca UK limited, Italy: jc393, 20 ml: 200 mg); 20% intralipid (2B6061; Baxter, Deerfield, IL, USA); 7,8‐Dihydroxyflavone (50 mg; Abcam, Cambridge, MA, USA); dimethyl sulphoxide (DMSO, 100 ml; Takara, Japan); TRIzol total RNA extraction kit (100 ml; Takara, Ohtsu, Shiga, Japan); RNAstore (100 ml; Takara); diethylpyrocarbonate (DEPC) (100 ml; Takara); MD101‐02 DNA Maker I (2 ml; Takara); Prime ScriptTM RT Reagent Kit with gDNA Eraser (Perfect Real Time) (Takara Code: RR047A for 100 * 20 μl reactions, Takara); SYBR Premix Ex TaqTM II (Tli RNaseH Plus) (Takara Code: RR820A for 200 * 50 μl reactions, Takara); BDNF, TrkB and β‐actin RT‐PCR primers (Sigma‐Aldrich, MA, USA); and DAB Colour Developing Reagent Kit for immunohistochemistry (IHC; DAKO, Carpinteria, CA, USA).

    Techniques: Expressing, Immunohistochemistry, Staining, Real-time Polymerase Chain Reaction